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Sin Gluten Salud

REFERENCIAS

Celiaquía

  1. Leffler DA, Green PH, Fasano A (octubre de 2015). «Extraintestinal manifestations of coeliac disease». Nat Rev Gastroenterol Hepatol 12 (10): 561-7. PMID 26260366. doi: 10.1038/nrgastro.2015.131. «Any organ from the central nervous system to joints, liver or teeth can be affected. Cualquier órgano, desde el sistema nervioso central hasta las articulaciones, el hígado o los dientes, puede verse afectado.».
  2. World Gastroenterology Organisation Global Guidelines (2016). «Celiac disease».
  3. Lundin KE, Wijmenga C (2015). «Coeliac disease and autoimmune disease-genetic overlap and screening». Nat Rev Gastroenterol Hepatol 12 (9): 507-15. PMID 26303674. doi:10.1038/nrgastro.2015.136.
  4. Ciccocioppo R, Kruzliak P, Cangemi GC, Pohanka M, Betti E, Lauret E, Rodrigo L (2015). «The Spectrum of Differences between Childhood and Adulthood Celiac Disease». Nutrients 7 (10): 8733-51. PMC 4632446. PMID 26506381. doi:10.3390/nu7105426.
  5. MedlinePlus. «Trastornos autoinmunitarios»
  6. National Institute of Environmental Health Sciences (NIEHS). «Autoimmune Diseases»
  7. Newnham ED (marzo de 2017). «Coeliac disease in the 21st century: paradigm shifts in the modern age». J Gastroenterol Hepatol . 32 Suppl 1: 82-85. PMID 28244672. doi:10.1111/jgh.13704.
  8. Polanco Allué, Isabel (2008). Ministerio de Sanidad y Consumo «Diagnóstico precoz de la enfermedad celíaca». Madrid, España.
  9. Molina-Infante J, Santolaria S, Sanders DS, Fernández-Bañares F (2015). «Systematic review: noncoeliac gluten sensitivity». Aliment Pharmacol Ther  41 (9): 807-20. PMID 25753138. doi:10.1111/apt.13155.
  10. Ludvigsson, JF; Card, T; Ciclitira, PJ; Swift, GL; Nasr, I; Sanders, DS; Ciacci, C (abril de 2015). «Support for patients with celiac disease: A literature review». United European Gastroenterol J 3 (2): 146-59. PMC 4406900. PMID 25922674. doi:10.1177/2050640614562599.
  11. Lionetti E, Gatti S, Pulvirenti A, Catassi C (2015). «Celiac disease from a global perspective». Best Pract Res Clin Gastroenterol.  29 (3): 365-79. PMID 26060103. doi:10.1016/j.bpg.2015.05.004
  12. Ludvigsson JF, Card TR, Kaukinen K, Bai J, Zingone F, Sanders DS, Murray JA. (2015). «Screening for celiac disease in the general population and in high-risk groups». United European Gastroenterol J 3 (2): 106-20. PMC 4406899. PMID 25922671. doi:10.1177/2050640614561668.
  13. Duggan JM (2004). «Coeliac disease: the great imitator». Med J Aust  180 (10): 524-6. PMID 1513983.
  14. Rousset H (2004). «A great imitator for the allergologist: intolerance to gluten» . Eur Ann Allergy Clin Immunol  36 (3): 96-100. PMID 15137480.
  15. Medeiros L (2006). «Celiac disease. A condition with multiple faces». Adv Nurse Pract  14 (4): 49-50, 52-4, 73. PMID 16972475.
  16.  Rodrigo, Luis; Garrote, José A.; Vivas, Santiago (2008). «Enfermedad Celíaca». Med Clin (Barc) 131 (7): 264-70. PMID 18775218.
  17. Green, PH (2005). «The many faces of celiac disease: clinical presentation of celiac disease in the adult population». Gastroenterology 128 (4 Suppl 1): S74-8. PMID 15825130. doi:10.1053/j.gastro.2005.02.016.
  18. Sanders, DS; Carter, MJ; Hurlstone, DP; Pearce, A; Ward, AM; McAlindon, ME (2001). «Association of adult coeliac disease with irritable bowel syndrome: a case-control study in patients fulfilling ROME IIcriteria referred to secondary care». Lancet 358 (9292): 1504-8. doi:10.1016/S0140-6736(01)06581-3.
  19. Sanders, DS; Hurlstone, DP; Stokes, RO; Rashid, F; Milford-Ward, A; Hadjivassiliou, M; et al. (2002). «Changing face of adult coeliac disease: experience of a single university hospital in South Yorkshire». Postgrad Med J 78 (915): 31-3. doi:10.1136/pmj.78.915.31.
  20. Green, PHR; Stavropoulos, SN; Panagi, SG; Goldstein, SL; Mcmahon, DJ; Absan, H; et al. (2001). «Characteristics of adult celiac disease in the USA: results of a national survey». Am J Gastroenterol 96: 126-31. doi:10.1111/j.1572-0241.2001.03462.x.
  21. Rodrigo, L (2010). «Enfermedad celiaca». IT Sistema Nacional Salud (Revisión) 34 (2): 52.
  22. «Symptoms & Causes of Celiac Disease What are the symptoms of celiac disease?». National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK).
  23. Moreno ML, Rodríguez-Herrera A, Sousa C, Comino I (enero de 2017). «Biomarkers to Monitor Gluten-Free Diet Compliance in Celiac Patients». Nutrients  6 (9): 1. PMC 5295090. PMID 28067823. doi:10.3390/nu9010046.
  24. See JA, Kaukinen K, Makharia GK, Gibson PR, Murray JA (2015). «Practical insights into gluten-free diets». Nat Rev Gastroenterol Hepatol 12 (10): 580-91. PMID 26392070. doi:10.1038/nrgastro.2015.156.
  25. Vriezinga SL, Schweizer JJ, Koning F, Mearin ML (2015). «Coeliac disease and gluten-related disorders in childhood». Nat Rev Gastroenterol Hepatol 12 (9): 527-36. PMID 26100369. doi:10.1038/nrgastro.2015.98.
  26. Dickey  W, Hughes DF, McMillan SA (2000). «Reliance on serum endomysial antibody testing underestimates the true prevalence of celiac disease by one fifth». Scand J Gastroenterol 35: 181-3.
  27. Han Y, Chen W, Li P, Ye J (2015). «Association Between Coeliac Disease and Risk of Any Malignancy and Gastrointestinal Malignancy: A Meta-Analysis.». Medicine (Baltimore) 94 (38): e1612. PMC 4635766. PMID 26402826. doi:10.1097/MD.0000000000001612.
  28. Woodward J (2016). «Improving outcomes of refractory celiac disease – current and emerging treatment strategies». Clinical and Experimental Gastroenterology 9: 225-36. PMC 4976763. PMID 27536154. doi:10.2147/ceg.s87200.
  29. Subprograma de Comercio al por menor e información al consumidor. Programa de Inspección y Apoyo al Control Oficial. Comunidad de Madrid. (2010). «Reacciones de hipersensibilidad a los alimentos. Normativa de aplicación en el control oficial de alérgenos presentes en los alimentos»

Celiaquía o Enfermedad celíaca: Diagnóstico. Criterios antiguos y criterios actuales.

  1. Rodrigo, Luis; Garrote, José A.; Vivas, Santiago (2008). «Enfermedad Celíaca». Med Clin (Barc) (131 (7): 264-70. PMID 18775218.
  2. Molina-Infante J, Santolaria S, Sanders DS, Fernández-Bañares F (2015). «Systematic review: noncoeliac gluten sensitivity». Aliment Pharmacol Ther 41 (9): 807-20. PMID 25753138. doi:10.1111/apt.13155.
  3. Rodrigo, L (2010). «Enfermedad celiaca». IT Sistema Nacional Salud (Revisión) 34 (2): 52.
  4. Duggan JM (2004). «Coeliac disease: the great imitator». Med J Aust 180 (10): 524-6. PMID 1513983.
  5. Rousset H (2004). «A great imitator for the allergologist: intolerance to gluten». Eur Ann Allergy Clin Immunol 36 (3): 96-100. PMID 15137480.
  6. Vriezinga SL, Schweizer JJ, Koning F, Mearin ML (2015). «Coeliac disease and gluten-related disorders in childhood». Nat Rev Gastroenterol Hepatol 12 (9): 527-36. PMID 26100369. doi:10.1038/nrgastro.2015.98.
  7. Catassi C, Fasano A (2010). «Celiac disease diagnosis: simple rules are better than complicated algorithms». The American Journal of Medicine 123 (8): 691–3.
  8. Lebwohl B, Rubio-Tapia A, Assiri A, Newland C, Guandalini S (2012). «Diagnosis of celiac disease». Gastrointest Endosc Clin N Am 22(4):661-77. PMID 23083985. doi:10.1016/j.giec.2012.07.004.
  9. Husby S, Koletzko S, Korponay-Szabó IR, Mearin ML, Phillips A, Shamir R, Troncone R, Giersiepen K, Branski D, Catassi C, Lelgeman M, Mäki M, Ribes-Koninckx C, Ventura A, Zimmer KP; ESPGHAN Working Group on Coeliac Disease Diagnosis; ESPGHAN Gastroenterology Committee; European Society for Pediatric Gastroenterology, Hepatology, and Nutrition (2012). «European Society for Pediatric Gastroenterology, Hepatology, and Nutrition guidelines for the diagnosis of coeliac disease». J Pediatr Gastroenterol Nutr 54(1):136-60. PMID 2197856. doi: 10.1097/MPG.0b013e31821a23d0.
  10. Leffler DA, Green PH, Fasano A (octubre de 2015). «Extraintestinal manifestations of coeliac disease». Nat Rev Gastroenterol Hepatol 12 (10): 561-7. PMID 26260366. doi: 10.1038/nrgastro.2015.131. «Any organ from the central nervous system to joints, liver or teeth can be affected. Cualquier órgano, desde el sistema nervioso central hasta las articulaciones, el hígado o los dientes, puede verse afectado.».
  11. Dickey, W; Hughes, DF; McMillan, SA (2000). «Reliance on serum endomysial antibody testing underestimates the true prevalence of celiac disease by one fifth». Scand J Gastroenterol 35: 181-3.
  12. Lewis, NR; Scott, BB (2006). «Systematic review: the use of serology to exclude or diagnose coeliac disease (a comparison of the endomysial and tissue transglutaminase antibody tests)». Aliment Pharmacol Ther 24: 47-54. PMID 16803602. doi:10.1111/j.1365-2036.2006.02967.x.
  13. Abrams, JA; Brar, P; Diamond, B; Rotterdam, H; Green, PH (2006). «Utility in clinical practice of immunoglobulin a anti-tissue transglutaminase antibody for the diagnosis of celiac disease». Clin Gastroenterol Hepatol 4: 726-30. PMID 16630760. doi:10.1016/j.cgh.2006.02.010.
  14. Ludvigsson, JF; Card, T; Ciclitira, PJ; Swift, GL; Nasr, I; Sanders, DS; Ciacci, C (abril de 2015). «Support for patients with celiac disease: A literature review». United European Gastroenterol J 3 (2): 146-59. PMC 4406900. PMID 25922674. doi:10.1177/2050640614562599.
  15. Newnham ED (marzo de 2017). «Coeliac disease in the 21st century: paradigm shifts in the modern age». J Gastroenterol Hepatol . 32 Suppl 1: 82-85. PMID 28244672. doi:10.1111/jgh.13704.
  16. López-Vázquez, A; Rodrigo, L; Fuentes, D; Riestra, S; Bousoño, C; García-Fernández, S; et al. (2002). «MHC class I chain related gene A (MICA) modulates the development of coeliac disease in patients with the high risk heterodimer DQA1*0501/DQB1*0201». Gut 50: 336-40. PMC 1773124. PMID 11839711. doi:10.1136/gut.50.3.336.
  17. Moreno ML, Rodríguez-Herrera A, Sousa C, Comino I (enero de 2017). «Biomarkers to Monitor Gluten-Free Diet Compliance in Celiac Patients». Nutrients  6 (9): 1. PMC 5295090. PMID 28067823. doi:10.3390/nu9010046.
  18. Castillo NE, Theethira TG, Leffler DA (2015). «The present and the future in the diagnosis and management of celiac disease». Gastroenterology Report 3 (1): 3–11. doi:10.1093/gastro/gou065. PMC 4324867. PMID 25326000.
  19. Levy J, Bernstein L, Silber N (2014). «Celiac disease: an immune dysregulation syndrome». Current Problems in Pediatric and Adolescent Health Care  44 (11): 324–7. doi:10.1016/j.cppeds.2014.10.002. PMID 25499458. “Initially, reduced levels of lactase and sucrase activities might necessitate further dietary restrictions until the villi have healed and those sugars are better tolerated.”
  20. Montalto M, Gallo A, Ojetti V, Gasbarrini A (2013). «Fructose, trehalose and sorbitol malabsorption». European Review for Medical and Pharmacological Sciences 17 Suppl 2 (Suppl 2): 26–9. PMID 24443064.
  21. Leffler DA, Green PH, Fasano A (octubre de 2015). «Extraintestinal manifestations of coeliac disease». Nat Rev Gastroenterol Hepatol 12 (10): 561-7. PMID 26260366. doi: 10.1038/nrgastro.2015.131. «Any organ from the central nervous system to joints, liver or teeth can be affected. Cualquier órgano, desde el sistema nervioso central hasta las articulaciones, el hígado o los dientes, puede verse afectado.».
  22. Woodward J (2016). «Improving outcomes of refractory celiac disease – current and emerging treatment strategies». Clinical and Experimental Gastroenterology 9: 225–36. doi:10.2147/ceg.s87200. PMC 4976763. PMID 27536154.

Sensibilidad al gluten

  1. Fasano A, Sapone A, Zevallos V, Schuppan D (mayo de 2015). «Nonceliac gluten sensitivity». Gastroenterology 148 (6): 1195-204. PMID 25583468. doi:10.1053/j.gastro.2014.12.049.
  2. Molina-Infante J, Santolaria S, Sanders DS, Fernández-Bañares F (2015). «Systematic review: noncoeliac gluten sensitivity». Aliment Pharmacol Ther 41 (9): 807-20. PMID 25753138. doi:10.1111/apt.13155.
  3. Mansueto P, Seidita A, D’Alcamo A, Carroccio A (2014). «Non-celiac gluten sensitivity: literature review». J Am Coll Nutr 33 (1): 39-54. PMID 24533607. doi:10.1080/07315724.2014.869996.
  4. Ludvigsson JF, Leffler DA, Bai JC, Biagi F, Fasano A, Green PH, Hadjivassiliou M, Kaukinen K, Kelly CP, Leonard JN, Lundin KE, Murray JA, Sanders DS, Walker MM, Zingone F, Ciacci C (2013). «The Oslo definitions for coeliac disease and related terms». Gut 62 (1): 43-52. PMC 3440559. PMID 22345659. doi:10.1136/gutjnl-2011-301346.
  5. Catassi C (2015). «Gluten Sensitivity». Ann Nutr Metab 67 (Suppl 2): 16-26. PMID 26605537. doi:10.1159/000440990.
  6. Elli L, Roncoroni L, Bardella MT (2015). «Non-celiac gluten sensitivity: Time for sifting the grain». World J Gastroenterol 21 (27): 8221-6. PMC 4507091. PMID 26217073. doi:10.3748/wjg.v21.i27.8221.
  7. Ontiveros N, Hardy MY, Cabrera-Chavez F (2015). «Assessing of Celiac Disease and Nonceliac Gluten Sensitivity». Gastroenterology Research and Practice 2015: 723954. PMC 4429206. PMID 26064097. doi:10.1155/2015/723954.
  8. Aziz I, Hadjivassiliou M, Sanders DS (2015). «The spectrum of noncoeliac gluten sensitivity». Nat Rev Gastroenterol Hepatol 12 (9): 516-26. PMID 26122473. doi:10.1038/nrgastro.2015.107.
  9. Catassi C, Bai JC, Bonaz B, Bouma G, Calabrò A, Carroccio A, Castillejo G, Ciacci C, Cristofori F, Dolinsek J, Francavilla R, Elli L, Green P, Holtmeier W, Koehler P, Koletzko S, Meinhold C, Sanders D, Schumann M, Schuppan D, Ullrich R, Vécsei A, Volta U, Zevallos V, Sapone A, Fasano A (2013). «Non-Celiac Gluten sensitivity: the new frontier of gluten related disorders». Nutrients 5 (10): 3839-53. PMC 3820047. PMID 24077239. doi:10.3390/nu5103839.
  10. Volta U, Caio G, Tovoli F, De Giorgio R (2013). «Non-celiac gluten sensitivity: questions still to be answered despite increasing awareness». Cellular and Molecular Immunology 10 (5): 383-392. ISSN 1672-7681. PMC 4003198. PMID 23934026. doi:10.1038/cmi.2013.28.
  11. Czaja-Bulsa G (2015). «Non coeliac gluten sensitivity – A new disease with gluten intolerance». Clin Nutr 34 (2): 189–94. PMID 25245857. doi:10.1016/j.clnu.2014.08.012.
  12. Verdu EF, Armstrong D, Murray JA (2009). «Between celiac disease and irritable bowel syndrome: the “no man’s land” of gluten sensitivity». Am J Gastroenterol 104 (6): 1587-94. PMC 3480312. PMID 19455131. doi:10.1038/ajg.2009.188.
  13. Picascia S, Mandile R, Auricchio R, Troncone R, Gianfrani C (2015). «Gliadin-Specific T-Cells Mobilized in the Peripheral Blood of Coeliac Patients by Short Oral Gluten Challenge: Clinical Applications». Nutrients 7 (12): 10020-31. PMID 26633487. doi:10.3390/nu7125515.
  14. Tonutti E, Bizzaro N (2014). «Diagnosis and classification of celiac disease and gluten sensitivity». Autoimmun Rev 13 (4-5): 472-6. PMID 24440147. doi:10.1016/j.autrev.2014.01.043.
  15. Fasano, A; Catassi, C (2012). «Clinical practice. Celiac disease». The New England Journal of Medicine 367 (25): 2419–26. PMID 23252527. doi:10.1056/NEJMcp1113994.
  16. Troncone, R; Jabri, B (2011). «Celiac disease and gluten-sensitivity». J Intern Med 269 (6): 582-90. PMID 21481018.
  17. Aziz, I; Sanders, DS (2012). «The irritable bowel syndrome-celiac disease connection». Gastrointest Endosc Clin N Am 22 (4): 623-37. PMID 23083983.
  18. Volta U, Caio G, De Giorgio R, Henriksen C, Skodje G, Lundin KE (2015). «Non-celiac gluten sensitivity: a work-in-progress entity in the spectrum of wheat-related disorders». Best Pract Res Clin Gastroenterol 29 (3): 477-91. PMID 26060112. doi:10.1016/j.bpg.2015.04.006.
  19. Jackson JR, Eaton WW, Cascella NG, Fasano A, Kelly DL (2012). «Neurologic and psychiatric manifestations of celiac disease and gluten sensitivity». Psychiatr Q 83 (1): 91-102. PMC 3641836. PMID 21877216. doi:10.1007/s11126-011-9186-y.
  20. Daulatzai MA (2015). «Non-celiac gluten sensitivity triggers gut dysbiosis, neuroinflammation, gut-brain axis dysfunction, and vulnerability for dementia». CNS Neurol Disord Drug Targets 14 (1): 110-31. PMID 25642988.
  21. Lebwohl B, Ludvigsson JF, Green PH (2015). «Celiac disease and non-celiac gluten sensitivity». BMJ 5: 351:h4347. PMC 4596973. PMID 26438584. doi:10.1136/bmj.h4347.
  22. Catassi C, Elli L, Bonaz B, Bouma G, Carroccio A, Castillejo G, Cellier C, Cristofori F, de Magistris L, Dolinsek J, Dieterich W, Francavilla R, Hadjivassiliou M, Holtmeier W, Körner U, Leffler DA, Lundin KE, Mazzarella G, Mulder CJ, Pellegrini N, Rostami K, Sanders D, Skodje GI, Schuppan D, Ullrich R, Volta U, Williams M, Zevallos VF, Zopf Y, Fasano A (2015). «Diagnosis of Non-Celiac Gluten Sensitivity (NCGS): The Salerno Experts’ Criteria». Nutrients 2015 Jun 18;7(6): 4966-77. PMC 4488826. PMID 26096570. doi:10.3390/nu7064966.
  23. Lundin, KEA; Alaedini, AA (2012). «Non-celiac gluten sensitivity». Gastrointest End Clin N Am 22 (4): 723-34. PMID 23083989.
  24. Porcelli B, Verdino V, Bossini L, Terzuoli L, Fagiolini A (2014). «Celiac and non-celiac gluten sensitivity: a review on the association with schizophrenia and mood disorders.». Auto Immun Highlights 5 (2): 55-61. PMC 4389040. PMID 26000156. doi:10.1007/s13317-014-0064-0.
  25. San Mauro I, Garicano E, Collado L, Ciudad MJ (2014). «Is gluten the great etiopathogenic agent of disease in the XXI century». Nutr Hosp 30 (6): 1203-10. PMID 25433099. doi:10.3305/nh.2014.30.6.7866.
  26. Rossi A, Di Lollo AC, Guzzo MP, Giacomelli C, Atzeni F, Bazzichi L, Di Franco M (2015). «Fibromyalgia and nutrition: what news?». Clin Exp Rheumatol 33 (1 Suppl 88): S117-25. PMID 25786053.
  27. Genuis SJ, Lobo RA (2014). «Gluten sensitivity presenting as a neuropsychiatric disorder». Gastroenterol Res Pract 2014: 293206. PMC 3944951. PMID 24693281. doi:10.1155/2014/293206.
  28. Losurdo G, Principi M, Iannone A, Amoruso A, Ierardi E, Di Leo A, Barone M (2018). «Extra-intestinal manifestations of non-celiac gluten sensitivity: An expanding paradigm». World J Gastroenterol 24 (14): 1521-1530. PMC 5897856. PMID 29662290. doi:10.3748/wjg.v24.i14.1521.
  29. Elli L, Branchi F, Tomba C, Villalta D, Norsa L, Ferretti F, Roncoroni L, Bardella MT (2015). «Diagnosis of gluten related disorders: Celiac disease, wheat allergy and non-celiac gluten sensitivity». World J Gastroenterol 21 (23): 7110-9. PMC 4476872. PMID 26109797. doi:10.3748/wjg.v21.i23.7110.
  30. Vriezinga SL, Schweizer JJ, Koning F, Mearin ML (2015). «Coeliac disease and gluten-related disorders in childhood». Nature Reviews. Gastroenterology & Hepatology 12 (9): 527-36. PMID 26100369. doi:10.1038/nrgastro.2015.98.
  31. Molina-Infante J, Santolaria S, Sanders DS, Fernández-Bañares F (2015). «Systematic review: noncoeliac gluten sensitivity». Aliment Pharmacol Ther 41 (9): 807-20. PMID 25753138. doi:10.1111/apt.13155.
  32. Schuppan D, Pickert G, Ashfaq-Khan M, Zevallos V (2015). «Non-celiac wheat sensitivity: differential diagnosis, triggers and implications». Best Pract Res Clin Gastroenterol 29 (3): 469-76. PMID 26060111. doi:10.1016/j.bpg.2015.04.002.
  33. Green PH, Lebwohl B, Greywoode R (2015). «Celiac disease». J Allergy Clin Immunol 135 (5): 1099-106. PMID 25956012. doi:10.1016/j.jaci.2015.01.044.
  34. Mariné, M; Fernández-Bañares, F; Alsina, M; et al. (2009). «Impact of mass screening for gluten-sensitive enteropathy in working population». World J Gastroenterol 15: 1331-8.
  35. Volta, U; Tovoli, F; Cicola, R; et al. (2012). «Serological tests in gluten sensitivity (nonceliac gluten intolerance)». J Clin Gastroenterol 46: 680-5.
  36. Sollid, LM; Lie, BA (2005). «Celiac disease genetics: current concepts and practical applications». Clin Gastroenterol Hepatol 3: 843–51.
  37. Lebwohl B, Rubio-Tapia A, Assiri A, Newland C, Guandalini S (2012). «Diagnosis of celiac disease». Gastrointest Endosc Clin N Am 22 (4): 661-77. PMC 4005880. PMID 23083985. doi:10.1016/j.giec.2012.07.004.

Enfermedades relacionadas con el gluten

  1. Biesiekierski JR (2017). «What is gluten?». J Gastroenterol Hepatol . 32 Suppl 1: 78-81. PMID 28244676. doi:10.1111/jgh.13703.
  2. San Mauro, Ismael; Garicano, E; Collado, L; Ciudad, MJ (2014). «Is gluten the great etiopathogenic agent of disease in the XXI century?». Nutr Hosp 30 (6): 1203-1210. PMID 25433099. doi:10.3305/nh.2014.30.6.7866.
  3. Bressan P, Kramer P (29 de marzo de 2016). «Bread and Other Edible Agents of Mental Disease». Front Hum Neurosci 10: 130. PMC 4809873. PMID 27065833. doi:10.3389/fnhum.2016.00130.
  4. See JA, Kaukinen K, Makharia GK, Gibson PR, Murray JA (2015). «Practical insights into gluten-free diets». Nat Rev Gastroenterol Hepatol 12 (10): 580-91. PMID 26392070. doi:10.1038/nrgastro.2015.156.
  5. Fasano A (abril de 2005). «Clinical presentation of celiac disease in the pediatric population». Gastroenterology 128 (4 Suppl 1): S68-73. PMID 15825129. doi:10.1053/j.gastro.2005.02.015.
  6. Lundin KE, Wijmenga C (septiembre de 2015). «Coeliac disease and autoimmune disease-genetic overlap and screening». Nat Rev Gastroenterol Hepatol 12 (9): 507-15. PMID 26303674. doi:10.1038/nrgastro.2015.136. «The abnormal immunological response elicited by gluten-derived proteins can lead to the production of several different autoantibodies, which affect different systems.».
  7. Lionetti E, Gatti S, Pulvirenti A, Catassi C (junio de 2015). «Celiac disease from a global perspective». Best Pract Res Clin Gastroenterol 29 (3): 365-79. PMID 26060103. doi:10.1016/j.bpg.2015.05.004.
  8. Verdu EF, Armstrong D, Murray JA (junio de 2009). «Between celiac disease and irritable bowel syndrome: the “no man’s land” of gluten sensitivity». Am J Gastroenterol 104 (6): 1587-94. PMC 3480312. PMID 19455131. doi:10.1038/ajg.2009.188.
  9. Mansueto P, Seidita A, D’Alcamo A, Carroccio A (2014). «Non-celiac gluten sensitivity: literature review». J Am Coll Nutr 33 (1): 39-54. PMID 24533607. doi:10.1080/07315724.2014.869996.
  10. Fedorak RN, Switzer CM, Bridges RJ (junio de 2012). «Canadian Digestive Health Foundation Public Impact Series 4: celiac disease in Canada. Incidence, prevalence, and direct and indirect economic impact». Can J Gastroenterol 26 (6): 350-2. PMC 3378282. PMID 22720277.
  11. Ludvigsson, JF; Card, T; Ciclitira, PJ; Swift, GL; Nasr, I; Sanders, DS; Ciacci, C (abril de 2015). «Support for patients with celiac disease: A literature review». United European Gastroenterol J 3 (2): 146-59. PMC 4406900. PMID 25922674. doi:10.1177/2050640614562599.
  12. Lionetti E, Gatti S, Pulvirenti A, Catassi C (junio de 2015). «Celiac disease from a global perspective». Best Pract Res Clin Gastroenterol. 29 (3): 365-79. PMID 26060103. doi:10.1016/j.bpg.2015.05.004.
  13. Ludvigsson JF, Card TR, Kaukinen K, Bai J, Zingone F, Sanders DS, Murray JA. (abril de 2015). «Screening for celiac disease in the general population and in high-risk groups». United European Gastroenterol J 3 (2): 106-20. PMC 4406899. PMID 25922671. doi:10.1177/2050640614561668.

Gluten: ¿qué significa?

  1. Miranda-Castro R, de-los-Santos-Álvarez N, Miranda-Ordieres AJ, Lobo-Castañón MJ (2016). «Harnessing Aptamers to Overcome Challenges in Gluten Detection ». Biosensors (Basel) 6 (2): 16. PMID 27104578. doi: 10.3390/bios6020016.
  2. El-Chammas K, Danner E (Jun 2011). «Gluten-free diet in nonceliac disease». Nutr Clin Pract  26 (3): 294-9. PMID 21586414. doi:10.1177/0884533611405538.
  3. Food and Drug Administration (January 2007). « Food Labeling ; Gluten-Free Labeling of Foods».

¿La avena tiene gluten? 2019: Explicación definitiva

  1. Biesiekierski JR (2017). «What is gluten?». J Gastroenterol Hepatol. 32 Suppl 1: 78-81. PMID 28244676. doi:10.1111/jgh.13703. “Similar proteins to the gliadin found in wheat exist as secalin in rye, hordein in barley, and avenins in oats and are collectively referred to as “gluten.” Derivatives of these grains such as triticale and malt and other ancient wheat varieties such as spelt and kamut also contain gluten. The gluten found in all of these grains has been identified as the component capable of triggering the immune-mediated disorder, coeliac disease. (Existen proteínas similares a la gliadina que se encuentra en el trigo, como las secalinas en el centeno, las hordeínas en la cebada y las aveninas en la avena, y se denominan colectivamente “gluten.” Derivados de estos granos, tales como el triticale y la malta, y otras variedades de trigo antiguas, tales como la espelta y el kamut, también contienen gluten. El gluten encontrado en todos estos granos ha sido identificado como el componente capaz de desencadenar el trastorno mediado por el sistema inmunitario, la enfermedad celíaca).”
  2. Food and Drug Administration (enero de 2007). «Food Labeling ; Gluten-Free Labeling of Foods». “There is no single definition of the term “gluten.” Technically, the term “gluten” refers to a specific complex of proteins that forms when wheat flour is mixed with a liquid and physically manipulated, such as in the kneading of a bread (Ref. 20). This complex of proteins is composed of both “gliadins” and “glutenins,” which are found in approximately equal proportions in most 11 wheat varieties (Refs. 21 through 23). The gliadins belong to a category of proteins called “prolamins” and the glutenins belong to a category of proteins called “glutelins” (Refs . 20 and 24). Although, strictly speaking, “gluten” pertains only to wheat proteins, this term is frequently used to refer to the combination of prolamin and glutelin proteins naturally occurring in other grains, including those that have not been demonstrated to cause harmful effects in individuals with celiac disease (e.g., “corn gluten” and “rice gluten”) (Ref. 25) . However, in discussions of celiac disease in the medical literature, the term “gluten” is used to refer to either gluten in wheat or collectively to the proteins (e.g., prolamins and glutelins) in just those grains that have been demonstrated to cause harmful health effects in individuals who have celiac disease (Refs . 3 and 25).
  3. Comino I, Real A, de Lorenzo L, Cornell H, López-Casado MÁ, Barro F, Lorite P, Torres MI, Cebolla A, Sousa C. «Diversity in oat potential immunogenicity: basis for the selection of oat varieties with no toxicity in coeliac disease». Gut. 2011 Jul;60(7):915-22. PMID  21317420. doi: 10.1136/gut.2010.225268.
  4. Lundin KE, Nilsen EM, Scott HG, Loberg EM, Gjoen A, Bratlie J, Skar V, Mendez E, Løvik A, Kett K (2003). «Oats induced villous atrophy in coeliac disease». Gut (2003) 52:1649–52. PMID 14570737. PMC 1773854. doi: 10.1136/gut.52.11.1649.
  5. Arentz-Hansen H, Fleckenstein B, Molberg O, Scott H, Koning F, Jung G, Roepstorff P, Lundin KE, Sollid LM (2004). «The molecular basis for oat intolerance in patients with celiac disease». PLoS Med. 1:e1. PMID 15526039. PMC 523824. doi:10.1371/journal.pmed.0010001.
  6. Hardy MY, Tye-Din JA, Stewart JA, Schmitz F, Dudek NL, Hanchapola I, Purcell AW, Anderson RP (2015). «Ingestion of oats and barley in patients with celiac disease mobilizes cross-reactive T cells activated by avenin peptides and immuno-dominant hordein peptides». J Autoimmun. 56:56–65. PMID 25457306. 10.1016/j.jaut.2014.10.003.
  7. Haboubi NY, Taylor S, Jones S (octubre de 2006). «Coeliac disease and oats: a systematic review». Postgrad Med J 82 (972): 672-8. PMC 2653911. PMID 17068278. doi:10.1136/pgmj.2006.045443.
  8. La Vieille, S; Pulido, O. M.; Abbott, M; Koerner, T. B.; Godefroy, S (2016). «Celiac Disease and Gluten-Free Oats: A Canadian Position Based on a Literature Review». Canadian Journal of Gastroenterology and Hepatology. 2016: 1–10. doi:10.1155/2016/1870305. PMC 4904695. PMID 27446825.
  9. de Souza MC, Deschênes ME, Laurencelle S, Godet P, Roy CC, Djilali-Saiah I (2016). «Pure Oats as Part of the Canadian Gluten-Free Diet in Celiac Disease: The Need to Revisit the Issue.». Can J Gastroenterol Hepatol  2016: 1576360. PMC 4904650. PMID 27446824. doi:10.1155/2016/1576360.
  10. Pinto-Sánchez MI, Causada-Calo N, Bercik P, Ford AC, Murray JA, Armstrong D, Semrad C, Kupfer SS, Alaedini A, Moayyedi P, Leffler DA, Verdú EF, Green P (agosto de 2017). «Safety of Adding Oats to a Gluten-Free Diet for Patients With Celiac Disease: Systematic Review and Meta-analysis of Clinical and Observational Studies». Gastroenterology. 153 (2): 395-409.e3. PMID 28431885. doi: 10.1053/j.gastro.2017.04.009.
  11. Comino I, Moreno M de L, Sousa C (7 Nov 2015). «Role of oats in celiac disease». World J Gastroenterol 21 (41): 11825-31. PMC 4631980. PMID 26557006. doi:10.3748/wjg.v21.i41.11825. «It is necessary to consider that oats include many varieties, containing various amino acid sequences and showing different immunoreactivities associated with toxic prolamins. As a result, several studies have shown that the immunogenicity of oats varies depending on the cultivar consumed. Thus, it is essential to thoroughly study the variety of oats used in a food ingredient before including it in a gluten-free diet.»
  12. Penagini F, Dilillo D, Meneghin F, Mameli C, Fabiano V, Zuccotti GV (18 Nov 18 2013). «Gluten-free diet in children: an approach to a nutritionally adequate and balanced diet». Nutrients 5 (11): 4553-65. PMC 3847748. PMID 24253052. doi:10.3390/nu5114553.
  13. Syage JA, Kelly CP, Dickason MA, Ramirez AC, Leon F, Dominguez R, Sealey-Voyksner JA (1 de febrero de 2018). «Determination of gluten consumption in celiac disease patients on a gluten-free diet». Am J Clin Nutr 107 (2): 201-207. PMID 29529159. doi:10.1093/ajcn/nqx049.
  14. Moreno ML, Rodríguez-Herrera A, Sousa C, Comino I (enero de 2017). «Biomarkers to Monitor Gluten-Free Diet Compliance in Celiac Patients». Nutrients  6 (9): 1. PMC 5295090. PMID 28067823. doi:10.3390/nu9010046.
  15. See JA, Kaukinen K, Makharia GK, Gibson PR, Murray JA (2015). «Practical insights into gluten-free diets». Nat Rev Gastroenterol Hepatol 12 (10): 580-91. PMID 26392070. doi:10.1038/nrgastro.2015.156. «A lack of symptoms and/or negative serological markers are not reliable indicators of mucosal response to the diet. La ausencia de síntomas y/o los marcadores serológicos negativos no son indicadores fiables de la respuesta de la mucosa intestinal a la dieta.»
  16. Newnham ED (marzo de 2017). «Coeliac disease in the 21st century: paradigm shifts in the modern age». J Gastroenterol Hepatol . 32 Suppl 1: 82-85. PMID 28244672. doi:10.1111/jgh.13704. «A significant weakness of all clinical studies in CD to date has been the absence of an objective measure of dietary compliance. Coeliac antibodies, intestinal histology, and symptoms are all imperfect tools for assessment of adherence. Una debilidad significativa de todos los estudios clínicos sobre enfermedad celíaca hasta la fecha ha sido la ausencia de una medida objetiva del cumplimiento dietético. Los anticuerpos celíacos, la histología intestinal y los síntomas son todos herramientas imperfectas para la evaluación de la adherencia.».
  17. Fasano A, Sapone A, Zevallos V, Schuppan D (mayo de 2015). «Nonceliac gluten sensitivity». Gastroenterology 148 (6): 1195-204. PMID 25583468. doi:10.1053/j.gastro.2014.12.049.
  18. «Agricultura ecológica en la UE: nuevas reglas más estrictas (infografía)». Noticias Parlamento Europeo. (10 de abril de 2018)
  19. Schalk K, Lexhaller B, Koehler P, Scherf KA (24 de febrero de 2017). «Isolation and characterization of gluten protein types from wheat, rye, barley and oats for use as reference materials». PLoS One. 12(2):e0172819. PMID 28234993. PMC 5325591. doi: 10.1371/journal.pone.0172819.

Gluten y Cerebro: cuando el gluten ataca al cerebro. Neurgluten.

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  2. Leffler DA, Green PH, Fasano A (octubre de 2015). «Extraintestinal manifestations of coeliac disease». Nat Rev Gastroenterol Hepatol 12 (10): 561-7. PMID 26260366. doi: 10.1038/nrgastro.2015.131. «Any organ from the central nervous system to joints, liver or teeth can be affected. Cualquier órgano, desde el sistema nervioso central hasta las articulaciones, el hígado o los dientes, puede verse afectado.».
  3. Bernier JJ, Buge A, Rambaud JC, Rancurel G, Hauw JJ, Modigliani R, Denvil D (octubre de 1976). «Nondeficiency chronic polyneuropathies in celiac disease in adults (2 cases with inflammatory neuromuscular vascularitis)». Ann Med Interne (Paris) (en francés) 127 (10): 721-9. PMID 1008365.
  4. Hadjivassiliou M, Sanders DD, Aeschlimann DP (2015). «Gluten-related disorders: gluten ataxia». Dig Dis 33 (2): 264-8. PMID 25925933. doi: 10.1159/000369509.
  5. Sapone, A; Bai, JC; Ciacci, C; Dolinsek, J; Verde, PH; Hadjivassiliou, M; et al. (2012). «Spectrum of gluten-related disorders: consensus on new nomenclature and classification». BMC Med 10: 13. PMC 329244. PMID 22313950. Ludvigsson, JF; Leffler, DA; Bai, JC; et al. (2013). «The Oslo definitions for coeliac disease and related terms». Gut 62 (1): 43-52. PMC 3440559. PMID 22345659. doi: 10.1136/gutjnl-2011-301346.
  6. Zis P, Hadjivassiliou M (26 de febrero de 2019). «Treatment of Neurological Manifestations of Gluten Sensitivity and Coeliac Disease». Curr Treat Options Neurol (Revisión) 21 (3): 10. PMID 30806821. doi: 10.1007/s11940-019-0552-7.
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  8. Lundin KE, Wijmenga C (Sep 2015). «Coeliac disease and autoimmune disease-genetic overlap and screening». Nat Rev Gastroenterol Hepatol12 (9): 507-15. PMID 26303674. doi: 10.1038/nrgastro.2015.136.
  9. San Mauro, Ismael; Garicano, E; Collado, L; Ciudad, MJ (2014). «Is gluten the great etiopathogenic agent of disease in the XXI century?» [¿Es el gluten el gran agente etiopatogénico de enfermedad en el siglo XXI?]. Nutr Hosp 30 (6): 1203-1210. PMID 25433099. doi: 10.3305/nh.2014.30.6.7866.
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  11. El-Chammas K, Danner E (Jun 2011). «Gluten-free diet in nonceliac disease». Nutr Clin Pract (Revisión) 26 (3): 294-9. PMID 21586414. doi: 10.1177/0884533611405538. «Historically, a GFD was occasionally used in the management of multiple sclerosis (MS), because anecdotal reports indicated a positive effect (reversal of symptoms) of a GFD in MS patients. (GFD: gluten-free diet). Históricamente, la DSG se ha utilizado ocasionalmente en el tratamiento de la esclerosis múltiple (EM), porque casos documentados indican un efecto positivo (reversión de los síntomas) de una DSG en pacientes con EM. DSG: dieta sin gluten».
  12. Hernández-Lahoz C, Rodrigo L (2013 Apr 15). «Gluten-related disorders and demyelinating diseases». Med Clin (Barc) (Revisión) 140 (7): 314-9. PMID22998972. doi: 10.1016/j.medcli.2012.07.009.
  13. Rosenbloom MH, Smith S, Akdal G, Geschwind MD (2009). «Immunologically mediated dementias». Curr Neurol Neurosci Rep (Revisión) 9 (5): 359-67. PMC 2832614. PMID 19664365. «Although most neurologists have experience diagnosing and treating typical dementias, such as those due to neurodegenerative conditions, including Alzheimer’s disease, few neurologists have as much familiarity with autoimmune causes of dementia. Whereas Alzheimer’s disease can be managed at a more leisurely pace, the immune-mediated dementias typically require urgent diagnosis and treatment with immunosuppressants or the underlying etiology. Increased awareness of the immune-mediated dementias and their comorbid symptoms should lead to prompt diagnosis and treatment of these fascinating and mysterious conditions.»
  14. Bushara KO (abril de 2005). «Neurologic presentation of celiac disease». Gastroenterology 128 (4 Suppl 1): S92-7. PMID 15825133.
  15. Makhlouf S, Messelmani M, Zaouali J, Mrissa R (marzo de 2018). «Cognitive impairment in celiac disease and non-celiac gluten sensitivity: review of literature on the main cognitive impairments, the imaging and the effect of gluten free diet». Acta Neurol Belg 118 (1): 21-27. PMID 29247390. doi: 10.1007/s13760-017-0870-z.
  16. Losurdo G, Principi M, Iannone A, Amoruso A, Ierardi E, Di Leo A, Barone M (14 de abril de 2018). «Extra-intestinal manifestations of non-celiac gluten sensitivity: An expanding paradigm». World J Gastroenterol 24 (14): 1521-1530. PMC5897856. PMID29662290. doi:10.3748/wjg.v24.i14.1521.
  17. Losurdo G, Principi M, Iannone A, Amoruso A, Ierardi E, Di Leo A, Barone M (14 de abril de 2018). «Extra-intestinal manifestations of non-celiac gluten sensitivity: An expanding paradigm». World J Gastroenterol 24 (14): 1521-1530. PMC 5897856. PMID 29662290. doi: 10.3748/wjg.v24.i14.1521.
  18. Baizabal-Carvallo JF, Jankovic J (julio de 2012). «Movement disorders in autoimmune diseases.». Mov Disord (Revisión) 27 (8): 935-46. PMID 22555904. doi: 10.1002/mds.25011.
  19. Catassi C, Bai JC, Bonaz B, Bouma G, Calabrò A, Carroccio A, Castillejo G, Ciacci C, Cristofori F, Dolinsek J, Francavilla R, Elli L, Green P, Holtmeier W, Koehler P, Koletzko S, Meinhold C, Sanders D, Schumann M, Schuppan D, Ullrich R, Vécsei A, Volta U, Zevallos V, Sapone A, Fasano A (Sep 2013). «Non-Celiac Gluten sensitivity: the new frontier of gluten related disorders». Nutrients (Revisión) 5 (10): 3839-53. PMC 3820047. PMID 24077239. doi: 10.3390/nu5103839.
  20. Kalaydjian, AE; Eaton, W; Cascella, N; Fasano, A (2006 Feb). «The gluten connection: the association between schizophrenia and celiac disease». Acta Psychiatr Scand113 (2): 82-90. PMID 16423158. doi: 10.1111/j.1600-0447.2005.00687.x.
  21. Volta U, Caio G, De Giorgio R, Henriksen C, Skodje G, Lundin KE (Jun 2015). «Non-celiac gluten sensitivity: a work-in-progress entity in the spectrum of wheat-related disorders». Best Pract Res Clin Gastroenterol 29 (3): 477-91. PMID 26060112. doi: 10.1016/j.bpg.2015.04.006.
  22. Buie T (2013). «The relationship of autism and gluten». Clin Ther 35 (5): 578–83. PMID 23688532. doi: 10.1016/j.clinthera.2013.04.011.
  23. Fasano A, Sapone A, Zevallos V, Schuppan D (May 2015). «Nonceliac gluten sensitivity». Gastroenterology 148 (6): 1195-204. PMID 25583468. doi:10.1053/j.gastro.2014.12.049.
  24. Couture DC, Chung MK, Shinnick P, Curzon J, McClure MJ, LaRiccia PJ (enero de 2016). «Integrative Medicine Approach to Pediatric Obsessive-Compulsive Disorder and Anxiety: A Case Report». Glob Adv Health Med5 (1): 117-21. PMC 4756770. PMID 26937323. doi: 10.7453/gahmj.2015.091.
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  28. Rodrigo L, Álvarez N, Fernández-Bustillo E, Salas-Puig J, Huerta M, Hernández-Lahoz C (7 de mayo de 2018). «Efficacy of a Gluten-Free Diet in the Gilles de la Tourette Syndrome: A Pilot Study.». Nutrients 10 (5): E573. PMID 29735930. doi: 10.3390/nu10050573.
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Autismo y gluten

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  2. Wasilewska J, Klukowski M (2015). “Gastrointestinal symptoms and autism spectrum disorder: links and risks – a possible new overlap syndrome”. Pediatric Health Med Ther. 6: 153–166. doi:10.2147/PHMT.S85717. PMC5 683266. PMID 29388597
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  6. Fasano, A (enero de 2011). «Zonulin and its regulation of intestinal barrier function: the biological door to inflammation, autoimmunity, and cancer». Physiol Rev 91 (1): 151-75. PMID 21248165. doi: 10.1152/physrev.00003.2008.
  7. Fasano, A (2012 Oct). «Intestinal permeability and its regulation by zonulin: diagnostic and therapeutic implications». Clin Gastroenterol Hepatol 10 (10): 1096-100. PMC 3458511. PMID 22902773. doi: 10.1016/j.cgh.2012.08.012.
  8. de Punder K1, Pruimboom L (12 de marzo de 2013). «The dietary intake of wheat and other cereal grains and their role in inflammation». Nutrients 5 (3): 771-87. PMC3705319. PMID23482055. doi:10.3390/nu5030771.
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  15. Christison GW, Ivany K (2006). «Elimination diets in autism spectrum disorders: any wheat amidst the chaff?». J Dev Behav Pediatr 27 (2 Suppl 2):S162–171. doi:10.1097/00004703-200604002-00015. PMID 16685183.
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  17. Buie T (2013). “The relationship of autism and gluten“. Clin Ther 35 (5): 578–83. doi:10.1016/j.clinthera.2013.04.011. PMID 23688532.
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  19. San Mauro I, Garicano E, Collado L, Ciudad MJ (December 2014). «¿Es el gluten el gran agente etiopatogenico de enfermedad en el siglo XXI? » [Is gluten the great etiopathogenic agent of disease in the XXI century?]. Nutr Hosp. 30 (6): 1203–10. doi:10.3305/nh.2014.30.6.7866. PMID 25433099.
  20. Leonard MM, Sapone A, Catassi C, Fasano A (2017). «Celiac Disease and Nonceliac Gluten Sensitivity: A Review». JAMA 318 (7): 647-656. doi:10.1001/jama.2017.9730. PMID 28810029.
  21. Lionetti E, Francavilla R, Pavone P, Pavone L, Francavilla T, Pulvirenti A, Giugno R, Ruggieri M (2010 Aug). «The neurology of coeliac disease in childhood: what is the evidence? A systematic review and meta-analysis». Dev Med Child Neurol 52 (8): 700-7. PMID 20345955. doi:10.1111/j.1469-8749.2010.03647.x.
  22. Hadjivassiliou M, Grünewald RA, Davies-Jones GA (mayo de 2002). «Gluten sensitivity as a neurological illness». J Neurol Neurosurg Psychiatry 72 (5): 560-3. PMC 1737870. PMID 11971034.
  23. World Gastroenterology Organisation Global Guidelines (julio de 2016). «Celiac disease».
  24. L. Alonso Canal, C. Isasi Zaragoza, I. Colmenero Blanco, M.J. Martínez Gómez, J. Arcas Martínez (18 de marzo de 2014). «Rasgos clínicos sugestivos de trastorno del espectro autista como manifestación de sensibilidad al gluten no celíaca». Anales de Pediatría 81(6):409-411

Nueva amenaza para los celiacos 2019

  1. «Logran avances en la resistencia de biocompuestos del trigo como alternativa a los plásticos contaminantes». (Agosto de 2018) Europa Press.
  2. «De vuelta al futuro: los científicos acuden a la naturaleza para buscar alternativas al plástico». (28 de noviembre de 2018) ONU Medio Ambiente.
  3. «Exploring the potential for adopting alternative materials to reduce marine plastic litter». (mayo de 2018) United Nations Environment Programme (UNEP).
  4. «Self-reinforced gluten polymers: A step towards a true biocomposite». (25 de abril de 2018) Open Access Government
  5. «New material gluten plastic used in sculpture at the Garden of Rosendal». (5 de julio de 2013) Innventia.com.
  6. Zink J, Wyrobnik T, Prinz T, Schmid M (23 de agosto de 2016). «Physical, Chemical and Biochemical Modifications of Protein-Based Films and Coatings: An Extensive Review». Int J Mol Sci 17 (9): pii: E1376. PMC 5037656. PMID 27563881. doi:10.3390/ijms17091376.
  7. Thakur R, Pristijono P, Scarlett CJ, Bowyer M, Singh SP, Vuong QV (marzo de 2019). «Starch-based films: Major factors affecting their properties». Int J Biol Macromol 132:1079-1089. doi: 10.1016/j.ijbiomac.2019.03.190. PMID 30926503. doi:10.1016/j.ijbiomac.2019.03.190.
  8. Wretfors C (2008). «Hemp fibre and reinforcements of wheat gluten plastics». Alnarp: Sveriges lantbruksuniv. ISBN 978-91-85911-79-0.
  9. Quintero CJ, Falguera V, Muñoz HA (2010). «Películas y recubrimientos comestibles: importancia y tendencias recientes en la cadena hortofrutícola». Revista Tumbaga (5): 93-118. ISSN 1909-4841.
  10. Day L, Augustin MA, Batey IL, Wrigley CW (2006). «Wheat-gluten uses and industry needs». Trends in Food Science & Technology 17 (2): 82- 90. doi:10.1016/j.tifs.2005.10.003.
  11. Molina-Infante J, Santolaria S, Sanders DS, Fernández-Bañares F (May 2015). «Systematic review: noncoeliac gluten sensitivity». Aliment Pharmacol Ther 41 (9): 807-20. PMID 25753138. doi:10.1111/apt.13155.
  12. Bressan P, Kramer P (29 de marzo de 2016). «Bread and Other Edible Agents of Mental Disease». Front Hum Neurosci 10: 130. PMC 4809873. PMID 27065833. doi:10.3389/fnhum.2016.00130.
  13. «Materiales en contacto con los alimentos». (9 de julio de 2019) Agencia Española de Consumo, Seguridad Alimentaria y Nutrición (AECOSAN)
  14. REGLAMENTO (CE) Nº 1935/2004 DEL PARLAMENTO EUROPEO Y DEL CONSEJO, de 27 de octubre de 2004, sobre los materiales y objetos destinados a entrar en contacto con alimentos y por el que se derogan las Directivas 80/590/CEE y 89/109/CEE.

Gluten inhalado: también te puede afectar

  1. Kasim S, Moriarty KJ, Liston R (junio de 2007). «Nonresponsive celiac disease due to inhaled gluten». N Engl J Med 356 (24): 2548-9. PMID 17568042.
  2. Robertson DA, Taylor N, Sidhu H, Britten A, Smith CL, Holdstock G (1989). Digestion 42 (2): 98-103. «Pulmonary permeability in coeliac disease and inflammatory bowel disease». PMID 2670648. doi: 10.1159/000199832.
  3. Leffler DA, Green PH, Fasano A (octubre de 2015). «Extraintestinal manifestations of coeliac disease». Nat Rev Gastroenterol Hepatol 12 (10): 561-7. PMID 26260366. doi: 10.1038/nrgastro.2015.131. «Any organ from the central nervous system to joints, liver or teeth can be affected. Cualquier órgano, desde el sistema nervioso central hasta las articulaciones, el hígado o los dientes, puede verse afectado.».
  4. Lundin KE, Wijmenga C (2015). «Coeliac disease and autoimmune disease-genetic overlap and screening». Nat Rev Gastroenterol Hepatol 12 (9): 507-15. PMID 26303674. doi:10.1038/nrgastro.2015.136.
  5. Khodadadi I1, Abdi M, Aliabadi M, Mirmoeini ES (2011). «Exposure to respirable flour dust and gliadin in wheat flour mills». J Occup Health. 2011;53(6): 417-422. PMID 21996928.
  6. Kolopp-Sarda MN, Massin N, Gobert B, Wild P, Moulin JJ, Béné MC, Faure GC (noviembre de 1994). «Humoral immune responses of workers occupationally exposed to wheat flour». Am J Ind Med 26(5): 671-679. PMID 7832214.
  7. Symptoms From Inhaling Airborne Gluten https://www.verywellhealth.com/suffering-symptoms-from-airborne-gluten-562332

Quinoa ¿tiene gluten?

  1. Organización de las Naciones Unidas para la Alimentación y la Agricultura (FAO). «Plataforma de información de la quinua. Propiedades nutricionales».
  2. Organización de las Naciones Unidas para la Alimentación y la Agricultura (FAO). «Quinua».
  3. Zevallos VF, Ellis HJ, Suligoj T, Herencia LI, Ciclitira PJ (agosto de 2012). «Variable activation of immune response by quinoa (Chenopodium quinoa Willd.) prolamins in celiac disease». Am J Clin Nutr. 96 (2): 337-344.PMID 22760575. doi: 10.3945/ajcn.111.030684.
  4. Zevallos VF, Herencia LI, Chang F, Donnelly S, Ellis HJ, Ciclitira PJ (febrero de 2014). «Gastrointestinal effects of eating quinoa (Chenopodium quinoa Willd.) in celiac patients». Am J Gastroenterol 109 (2): 270-278. PMID 24445568. doi:10.1038/ajg.2013.431.
  5. Peñas E, Uberti F, di Lorenzo C, Ballabio C, Brandolini A, Restani P (diciembre de 2014). «Biochemical and immunochemical evidences supporting the inclusion of quinoa (Chenopodium quinoa Willd.) as a gluten-free ingredient». Plant Foods Hum Nutr. 69 (4): 297-303. PMID 25359556. doi: 10.1007/s11130-014-0449-2.
  6. Miguel MG (abril de 2018). «Betalains in Some Species of the Amaranthaceae Family: A Review». Antioxidants (Basel). 7 (4). pii: E53. PMID 29617324. PMCID 5946119. doi:10.3390/antiox7040053.
  7. Stoven S, Murray JA, Marietta EV (abril de 2013). «Latest in vitro and in vivo models of celiac disease». Expert Opin Drug Discov. 8 (4): 445-457. PMID 23293929. PMC 3605231. doi:10.1517/17460441.2013.761203.
  8. Comino I, Moreno M de L, Real A, Rodríguez-Herrera A, Barro F, Sousa C (23 de octubre de 2013). «The gluten-free diet: testing alternative cereals tolerated by celiac patients». Nutrients 5 (10): 4250-4268. PMID 24152755. PMCID 3820072. doi: 10.3390/nu5104250.
  9. Moreno ML, Rodríguez-Herrera A, Sousa C, Comino I (enero de 2017). «Biomarkers to Monitor Gluten-Free Diet Compliance in Celiac Patients». Nutrients (Revisión) 6 (9): 1. PMID 28067823. PMCID 5295090. doi: 10.3390/nu9010046.
  10. See JA, Kaukinen K, Makharia GK, Gibson PR, Murray JA (octubre de 2015). «Practical insights into gluten-free diets». Nat Rev Gastroenterol Hepatol (Review) 12 (10): 580-91. PMID 26392070. doi: 10.1038/nrgastro.2015.156.

¿Crees que haces bien la dieta sin gluten? ¿Cómo saber si te estás contaminando?

  1. Comino, I; Real, A; Moreno, ML; Cebolla, A; Sousa, C (2013). «Detección de la fracción inmunotóxica del gluten: aplicaciones en seguridad alimentaria y en la monitorización de pacientes celíacos». Enfermedad celíaca y sensibilidad al gluten no celíaca: 433-445. doi:10.3926/oms.24
  2. Allen KJ, Turner PJ, Pawankar R, Taylor S, Sicherer S, Lack G, Rosario N, Ebisawa M, Wong G, Mills EN, Beyer K, Fiocchi A, Sampson HA (2014). «Precautionary labelling of foods for allergen content: are we ready for a global framework?». World Allergy Organ J. 7 (1): 1–14. doi:10.1186/1939-4551-7-10. PMC 4005619. PMID 24791183.
  3. Hadjivassiliou M, Grünewald RA, Davies-Jones GA (mayo de 2002). «Gluten sensitivity as a neurological illness». J Neurol Neurosurg Psychiatry 72 (5): 560-3. PMC 1737870. PMID 11971034.
  4. See JA, Kaukinen K, Makharia GK, Gibson PR, Murray JA (2015). «Practical insights into gluten-free diets». Nat Rev Gastroenterol Hepatol 12 (10): 580-91. PMID 26392070. doi:10.1038/nrgastro.2015.156. «Interestingly, several studies show incomplete histological normalization of small bowel mucosa despite a strict GFD, and even 79% of the treated patients have persistent villous atrophy. This finding could be explained by an inadvertent gluten intake. Curiosamente, varios estudios muestran normalización histológica incompleta de la mucosa del intestino delgado a pesar de una estricta GFD, e incluso el 79% de los pacientes tratados tienen atrofia vellositaria persistente. Este hallazgo podría explicarse por una ingesta inadvertida de gluten.».
  5. Ministerio de Sanidad, Servicios Sociales e Igualdad de España (2018). «Protocolo para el diagnóstico precoz de la enfermedad celíaca».
  6. Lundin KE, Wijmenga C (septiembre de 2015). «Coeliac disease and autoimmune disease-genetic overlap and screening». Nat Rev Gastroenterol Hepatol 12 (9): 507-15. PMID 26303674. doi:10.1038/nrgastro.2015.136. «The abnormal immunological response elicited by gluten-derived proteins can lead to the production of several different autoantibodies, which affect different systems.».
  7. Han Y, Chen W, Li P, Ye J (2015). «Association Between Coeliac Disease and Risk of Any Malignancy and Gastrointestinal Malignancy: A Meta-Analysis.». Medicine (Baltimore) 94 (38): e1612. PMC 4635766. PMID 26402826. doi:10.1097/MD.0000000000001612.
  8. Ciaccio EJ, Lewis SK, Biviano AB, Iyer V, Garan H, Green PH (26 de agosto de 2017). «Cardiovascular involvement in celiac disease». World J Cardiol 9 (8): 652-666. PMC 5583538. PMID 28932354. doi:10.4330/wjc.v9.i8.652.
  9. Hourigan CS (junio de 2006). «The molecular basis of coeliac disease». Clin Exp Med 6 (2): 53-9. PMID 16820991.
  10. Nadhem ON, Azeez G, Smalligan RD, Urban S (abril de 2015). «Review and practice guidelines for celiac disease in 2014». Postgrad Med 127 (3): 259-65. PMID 25702766. doi:10.1080/00325481.2015.1015926.
  11. Green, PH; Jabri, B (agosto de 2003). «Coeliac disease». Lancet 362 (9381): 383-91. PMID 12907013.
  12. Woodward J (3 de agosto de 2016). «Improving outcomes of refractory celiac disease – current and emerging treatment strategies». Clinical and experimental gastroenterology 9: 225-36. PMC 4976763. PMID 27536154. doi:10.2147/ceg.s87200.
  13. Hadjivassiliou M, Duker AP, Sanders DS (2014). «Gluten-related neurologic dysfunction». Handb Clin Neurol 120: 607-19. PMID 24365341. doi:10.1016/B978-0-7020-4087-0.00041-3.
  14. Fasano A, Catassi C (20 de diciembre de 2012). «Clinical practice. Celiac disease». N Engl J Med 367 (25): 2419-26. PMID 23252527. doi:10.1056/NEJMcp1113994. «celiac crisis, a rare life-threatening syndrome, mostly observed in children, that is characterized by severe diarrhea, hypoproteinemia, and metabolic and electrolyte imbalances. crisis celíaca, un síndrome raro que pone en peligro la vida, principalmente observado en niños, que se caracteriza por diarrea severa, hipoproteinemia y desequilibrios metabólicos y electrolíticos.”».
  15. de Almeida Menezes M, Cabral V, Silva Lorena SL (enero de 2017). «Celiac crisis in adults: a case report and review of the literature focusing in the prevention of refeeding syndrome». Rev Esp Enferm Dig 109 (1): 67-68. PMID26912167. doi:10.17235/reed.2016.4073/2015.
  16. Ruiz, A; Polanco, I (2002). «Exposición al gluten y aparición de enfermedades autoinmunes en la enfermedad celíaca». Pediátrika 22: 311-9.
  17. Mulder CJ, van Wanrooij RL, Bakker SF, Wierdsma N, Bouma G (2013). «Gluten-free diet in gluten-related disorders». Dig Dis 31 (1): 57-62. PMID 23797124. doi:10.1159/000347180.
  18. See JA, Kaukinen K, Makharia GK, Gibson PR, Murray JA (2015). «Practical insights into gluten-free diets». Nat Rev Gastroenterol Hepatol 12 (10): 580-91. PMID 26392070. doi:10.1038/nrgastro.2015.156. «A lack of symptoms and/or negative serological markers are not reliable indicators of mucosal response to the diet. La ausencia de síntomas y/o los marcadores serológicos negativos no son indicadores fiables de la respuesta de la mucosa intestinal a la dieta.».
  19. Newnham ED (marzo de 2017). «Coeliac disease in the 21st century: paradigm shifts in the modern age». J Gastroenterol Hepatol . 32 Suppl 1: 82-85. PMID 28244672. doi:10.1111/jgh.13704. «A significant weakness of all clinical studies in CD to date has been the absence of an objective measure of dietary compliance. Coeliac antibodies, intestinal histology, and symptoms are all imperfect tools for assessment of adherence. Una debilidad significativa de todos los estudios clínicos sobre enfermedad celíaca hasta la fecha ha sido la ausencia de una medida objetiva del cumplimiento dietético. Los anticuerpos celíacos, la histología intestinal y los síntomas son todos herramientas imperfectas para la evaluación de la adherencia.».
  20. Ludvigsson, JF; Card, T; Ciclitira, PJ; Swift, GL; Nasr, I; Sanders, DS; Ciacci, C (abril de 2015). «Support for patients with celiac disease: A literature review». United European Gastroenterol J 3 (2): 146-59. PMC 4406900. PMID 25922674. doi:10.1177/2050640614562599.
  21. Schuppan D, Pickert G, Ashfaq-Khan M, Zevallos V (Jun 2015). «Non-celiac wheat sensitivity: differential diagnosis, triggers and implications». Best Pract Res Clin Gastroenterol 29 (3): 469-76. PMID 26060111. doi:10.1016/j.bpg.2015.04.002.
  22. Moreno ML, Rodríguez-Herrera A, Sousa C, Comino I (enero de 2017). «Biomarkers to Monitor Gluten-Free Diet Compliance in Celiac Patients». Nutrients 6 (9): 1. PMC 5295090. PMID 28067823. doi:10.3390/nu9010046.
  23. Lewis NR, Scott BB (1 de julio de 2006). «Systematic review: the use of serology to exclude or diagnose coeliac disease (a comparison of the endomysial and tissue transglutaminase antibody tests)». Aliment Pharmacol Ther (Revisión) 24 (1): 47-54. PMID 16803602. doi:10.1111/j.1365-2036.2006.02967.x.
  24. Abrams, JA; Brar, P; Diamond, B; Rotterdam, H; Green, PH (2006). «Utility in clinical practice of immunoglobulin a anti-tissue transglutaminase antibody for the diagnosis of celiac disease». Clin Gastroenterol Hepatol 4: 726-30. PMID 16630760. doi:10.1016/j.cgh.2006.02.010.
  25. Dickey, W; Hughes, DF; McMillan, SA (2000). «Reliance on serum endomysial antibody testing underestimates the true prevalence of celiac disease by one fifth». Scand J Gastroenterol 35: 181-3.
  26. Rodrigo, L (2010). «Enfermedad celiaca». IT Sistema Nacional Salud (Revisión) 34 (2): 52.
  27. Ministerio de Sanidad, Servicios Sociales e Igualdad de España (2018). «Protocolo para el diagnóstico precoz de la enfermedad celíaca».
  28. Lammers KM, Herrera MG, Dodero VI (27 de febrero de 2018). «Translational Chemistry Meets Gluten-Related Disorders». ChemistryOpen 7 (3): 217-232. PMC 5838388. PMID 29531885. doi:10.1002/open.201700197.
  29. Ludvigsson JF, Card TR, Kaukinen K, Bai J, Zingone F, Sanders DS, Murray JA. (abril de 2015). «Screening for celiac disease in the general population and in high-risk groups». United European Gastroenterol J 3 (2): 106-20. PMC 4406899. PMID 25922671. doi:10.1177/2050640614561668.
  30. Elli L, Villalta D, Roncoroni L, Barisani D, Ferrero S, Pellegrini N, Bardella MT, Valiante F, Tomba C, Carroccio A, Bellini M, Soncini M, Cannizzaro R, Leandro G (febrero de 2017). «Nomenclature and diagnosis of gluten-related disorders: A position statement by the Italian Association of Hospital Gastroenterologists and Endoscopists (AIGO)». Dig Liver Dis 49 (2): 138-146. PMID 27887897.
  31. Syage JA, Kelly CP, Dickason MA, Ramirez AC, Leon F, Dominguez R, Sealey-Voyksner JA (1 de febrero de 2018). «Determination of gluten consumption in celiac disease patients on a gluten-free diet». Am J Clin Nutr 107 (2): 201-207. PMID 29529159. doi:10.1093/ajcn/nqx049.
  32. «Dra. Moreno: “El test de detección de gluten -GlutenDetect- permite saber si se producen ingestas puntuales de la proteína”». Celicidad. 2 de abril de 2017.

Demencia por gluten

  1. «Demencia».  MedlinePlus. Enciclopedia médica.
  2. Zis P, Hadjivassiliou M (26 de febrero de 2019). «Treatment of Neurological Manifestations of Gluten Sensitivity and Coeliac Disease». Curr Treat Options Neurol 21 (3): 10. PMID 30806821. doi:10.1007/s11940-019-0552-7.
  3. Makhlouf S, Messelmani M, Zaouali J, Mrissa R (2018). «Cognitive impairment in celiac disease and non-celiac gluten sensitivity: review of literature on the main cognitive impairments, the imaging and the effect of gluten free diet». Acta Neurol Belg 118 (1): 21–27.
  4. Rosenbloom MH, Smith S, Akdal G, Geschwind MD (2009). «Immunologically mediated dementias». Curr Neurol Neurosci Rep 9 (5): 359-67. PMC 2832614. PMID 19664365. «Although most neurologists have experience diagnosing and treating typical dementias, such as those due to neurodegenerative conditions, including Alzheimer’s disease, few neurologists have as much familiarity with autoimmune causes of dementia. Whereas Alzheimer’s disease can be managed at a more leisurely pace, the immune-mediated dementias typically require urgent diagnosis and treatment with immunosuppressants or the underlying etiology. Increased awareness of the immune-mediated dementias and their comorbid symptoms should lead to prompt diagnosis and treatment of these fascinating and mysterious conditions.»
  5. Bushara KO (abril de 2005). «Neurologic presentation of celiac disease». Gastroenterology 128 (4 Suppl 1): S92-7. PMID 15825133
  6. Hadjivassiliou M, Grünewald RA, Davies-Jones GA (mayo de 2002). «Gluten sensitivity as a neurological illness». J Neurol Neurosurg Psychiatry 72 (5): 560-3. PMC 1737870. PMID 11971034.
  7. Ciacci C, Ciclitira P, Hadjivassiliou M, Kaukinen K, Ludvigsson JF, McGough N, Sanders DS, Woodward J, Leonard JN, Swift GL (abril de 2015). «The gluten-free diet and its current application in coeliac disease and dermatitis herpetiformis». United European Gastroenterol J 3 (2): 121-35. PMC 4406897. PMID 25922672. doi:10.1177/2050640614559263.
  8. CODEX STAN 118-2008. Standard for Foods for Special Dietary Use for Persons Intolerant to Gluten.
  9. Moreno ML, Rodríguez-Herrera A, Sousa C, Comino I (enero de 2017). «Biomarkers to Monitor Gluten-Free Diet Compliance in Celiac Patients». Nutrients  6 (9): 1. PMC 5295090. PMID 28067823. doi:10.3390/nu9010046.
  10. Leffler DA, Green PH, Fasano A (octubre de 2015). «Extraintestinal manifestations of coeliac disease». Nat Rev Gastroenterol Hepatol 12 (10): 561-7. PMID 26260366. doi: 10.1038/nrgastro.2015.131. «Any organ from the central nervous system to joints, liver or teeth can be affected. Cualquier órgano, desde el sistema nervioso central hasta las articulaciones, el hígado o los dientes, puede verse afectado.».
  11. World Gastroenterology Organisation Global Guidelines (julio de 2016). «Celiac disease». «The most important obstacle to implementing the recommendations is poor awareness of celiac disease by patients and physicians. Among gastroenterologists, there is a sense that celiac disease does not require follow-up by a specialized physician after diagnosis and may be considered a minor condition in comparison with irritable bowel disease (IBD) and inflammatory bowel syndrome (IBS).(…) There is an urgent need to increase awareness among primary-care physicians and pediatricians about the wide diversity of clinical manifestations. El obstáculo más importante para implementar las recomendaciones es el pobre conocimiento de la enfermedad celíaca por parte de los pacientes y los médicos. Entre los gastroenterólogos, existe la sensación de que la enfermedad celíaca no requiere el seguimiento por un médico especialista después del diagnóstico y que puede considerarse una condición menor en comparación con el síndrome del intestino irritable (SII) y lA enfermedad inflamatoria intestinal(EII).(…) Existe una necesidad urgente de aumentar la conciencia entre los médicos de atención primaria y los pediatras acerca de la gran diversidad de manifestaciones clínicas».
  12. Lundin KE, Wijmenga C (septiembre de 2015). «Coeliac disease and autoimmune disease-genetic overlap and screening». Nat Rev Gastroenterol Hepatol 12 (9): 507-15. PMID 26303674. doi:10.1038/nrgastro.2015.136. «The abnormal immunological response elicited by gluten-derived proteins can lead to the production of several different autoantibodies, which affect different systems.».
  13. Ministerio de Sanidad, Servicios Sociales e Igualdad de España (2018). «Protocolo para el diagnóstico precoz de la enfermedad celíaca».
  14. Fasano A, Sapone A, Zevallos V, Schuppan D (May 2015). «Nonceliac gluten sensitivity». Gastroenterology 148 (6): 1195-204. PMID 25583468. doi:10.1053/j.gastro.2014.12.049.
  15. Herald Sun (18 de noviembre de 2014). «An increasing number of Australians are choosing a gluten-free diet».
  16. Nash DT, Slutzky AR (octubre de 2014). «Gluten sensitivity: new epidemic or new myth?». Proc (Bayl Univ Med Cent) 27 (4): 377-8. PMC 4255872. PMID 25484517.